Fellows' Publications
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Chronic Activation of γ2 AMPK Induces Obesity and Reduces β Cell Function.
Yavari A. et al, (2016), Cell Metab, 23, 821 - 836
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Insights into islet development and biology through characterization of a human iPSC-derived endocrine pancreas model.
van de Bunt M. et al, (2016), Islets, 8, 83 - 95
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Hyperglycaemia induces metabolic dysfunction and glycogen accumulation in pancreatic β-cells.
Brereton MF. et al, (2016), Nat Commun, 7
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Transcript Expression Data from Human Islets Links Regulatory Signals from Genome-Wide Association Studies for Type 2 Diabetes and Glycemic Traits to Their Downstream Effectors.
van de Bunt M. et al, (2015), PLoS Genet, 11
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A Low-Frequency InactivatingAKT2Variant Enriched in the Finnish Population Is Associated With Fasting Insulin Levels and Type 2 Diabetes Risk.
Manning A. et al, (2017), Diabetes, 66, 2019 - 2032
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Integrative genomic analysis implicates limited peripheral adipose storage capacity in the pathogenesis of human insulin resistance.
Lotta LA. et al, (2017), Nat Genet, 49, 17 - 26
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Systematic Functional Characterization of Candidate Causal Genes for Type 2 Diabetes Risk Variants.
Thomsen SK. et al, (2016), Diabetes, 65, 3805 - 3811
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The genetic architecture of type 2 diabetes.
Fuchsberger C. et al, Nature, 536, 41 - 47
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Optimizing human hepatocyte models for metabolic phenotype and function: effects of treatment with dimethyl sulfoxide (DMSO).
Nikolaou N. et al, (2016), Physiol Rep, 4
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The role of PYY in pancreatic islet physiology and surgical control of diabetes
Guida C. et al, Trends in Endocrinology and Metabolism