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DNA from individuals heterozygous for (G)gamma(deltabeta)(o) thalassaemia has been studied by restriction endonuclease analysis. The results reveal a new molecular defect associated with this condition. A total of three defects is now responsible for the one single phenotype, thereby emphasising the complex relationship between genotype and phenotype among the disorders of beta-like globin synthesis in man.

Type

Journal article

Journal

Nucleic Acids Res

Publisher

Oxford University Press (OUP): Policy C - Option B

Volume

9

Pages

6813 - 25