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Persistent hyperinsulinemic hypoglycemia of infancy (PHHI) is a disorder of childhood associated with inappropriate hypersecretion of insulin by the pancreas. The pathogenesis of the condition has hitherto remained controversial. We show here that insulin-secreting cells from a homogeneous group of five infants with PHHI lack ATP-sensitive K+ channel (KATP) activity. As a consequence, PHHI beta-cells are spontaneously electrically active with high basal cytosolic Ca2+ concentrations due to Ca2+ influx. Our findings define the pathogenesis of this disease as a novel K+ channel disorder.

Original publication

DOI

10.1038/nm1296-1344

Type

Journal article

Journal

Nature medicine

Publication Date

12/1996

Volume

2

Pages

1344 - 1347

Addresses

Department of Biomedical Science, University of Sheffield, UK.

Keywords

Islets of Langerhans, Cells, Cultured, Humans, Hyperinsulinism, Hypoglycemia, Verapamil, ATP-Binding Cassette Transporters, Potassium Channels, Potassium Channels, Inwardly Rectifying, Receptors, Drug, Adenosine Triphosphate, Calcium Channel Blockers, Action Potentials, Infant, Newborn, Sulfonylurea Receptors