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A recent study has revealed that loss of neuronal expression of the paternally imprinted gene Ube3a in Angelman syndrome results in selective neuronal loss of robust circadian oscillations, with a resulting behavioural phenotype, and adipose tissue accumulation.

Original publication

DOI

10.1016/j.cub.2014.12.045

Type

Journal article

Journal

Curr Biol

Publication Date

02/03/2015

Volume

25

Pages

R201 - R203

Keywords

Angelman Syndrome, Animals, Circadian Rhythm, Genomic Imprinting, Humans, Neurons, Ubiquitin-Protein Ligases