Publications — Radcliffe Department of Medicine

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Deciphering signaling pathways in hematopoietic stem cells: the molecular complexity of Myelodysplastic Syndromes (MDS) and leukemic progression.

Casalin I. et al, (2024), Adv Biol Regul

Classification, risk stratification and response assessment in myelodysplastic syndromes/neoplasms (MDS): A state-of-the-art report on behalf of the International Consortium for MDS (icMDS).

Stahl M. et al, (2023), Blood Rev, 62

Recent advances in the application of induced pluripotent stem cell technology to the study of myeloid malignancies

Tatwavedi D. et al, (2023), Advances in Biological Regulation, 100993 - 100993

Current landscape of translational and clinical research in myelodysplastic syndromes/neoplasms (MDS): Proceedings from the 1st International Workshop on MDS (iwMDS) Of the International Consortium for MDS (icMDS).

Bewersdorf JP. et al, (2023), Blood Rev, 60

A miRNA screening identifies miR-192-5p as associated with response to azacitidine and lenalidomide therapy in myelodysplastic syndromes.

Mongiorgi S. et al, (2023), Clin Epigenetics, 15

Whole-genome sequencing of chronic lymphocytic leukemia identifies subgroups with distinct biological and clinical features.

Robbe P. et al, (2022), Nat Genet, 54, 1675 - 1689

Finding consistency in classifications of myeloid neoplasms: a perspective on behalf of the International Workshop for Myelodysplastic Syndromes.

Zeidan AM. et al, (2022), Leukemia

An agenda to advance research in MDS: A TOP 10 Priority List from the first international workshop in MDS (iwMDS).

Stahl M. et al, (2022), Blood Adv

Splicing factor mutations in the myelodysplastic syndromes: Role of key aberrantly spliced genes in disease pathophysiology and treatment.

Pellagatti A. and Boultwood J., (2022), Adv Biol Regul

Activation of targetable inflammatory immune signaling is seen in myelodysplastic syndromes with SF3B1 mutations.

Choudhary GS. et al, (2022), Elife, 11

Pathophysiologic and clinical implications of molecular profiles resultant from deletion 5q.

Adema V. et al, (2022), EBioMedicine, 80

BCL-2 Inhibitor ABT-737 Effectively Targets Leukemia-Initiating Cells with Differential Regulation of Relevant Genes Leading to Extended Survival in a NRAS/BCL-2 Mouse Model of High Risk-Myelodysplastic Syndrome.

Gorombei P. et al, (2021), Int J Mol Sci, 22

ASXL1 mutations are associated with distinct epigenomic alterations that lead to sensitivity to venetoclax and azacytidine

Rahmani NE. et al, (2021), Blood Cancer Journal, 11

MDS DISEASE PROGRESSION: INSIGHTS FROM THE TRANSCRIPTOME

Boultwood J., (2021), LEUKEMIA RESEARCH, 108, S56 - S56

Author Correction: Implications of TP53 allelic state for genome stability, clinical presentation and outcomes in myelodysplastic syndromes.

Bernard E. et al, (2021), Nat Med, 27

MDMX acts as a pervasive preleukemic-to-acute myeloid leukemia transition mechanism.

Ueda K. et al, (2021), Cancer Cell

Author Correction: Implications of TP53 allelic state for genome stability, clinical presentation and outcomes in myelodysplastic syndromes.

Bernard E. et al, (2021), Nat Med, 27

The utilisation of glutamine and glucose by a 3-D tumour model trapped in quiescence.

Smith H. et al, (2021), Int J Biochem Cell Biol

SF3B1 mutant myelodysplastic syndrome: Recent advances.

Pellagatti A. and Boultwood J., (2020), Adv Biol Regul

Author Correction: Retrospective evaluation of whole exome and genome mutation calls in 746 cancer samples.

Bailey MH. et al, (2020), Nat Commun, 11

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