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Effects of canagliflozin on human myocardial redox signalling: clinical implications

Journal article

Kondo H. et al, (2021), European Heart Journal

Brown Adipose Tissue and the Take (12,13-di)HOME Message to the Heart.

Journal article

Badi I. and Antoniades C., (2021), Circulation, 143, 160 - 162

Direct effects of canagliflozin on human myocardial redox signalling: a novel role for SGLT1 inhibition

Conference paper

Kondo H. et al, (2020), EUROPEAN HEART JOURNAL, 41, 3351 - 3351

Role of HMGB1 in Vascular Smooth Muscle Cells (VSMCs) senescence and calcification

Conference paper

Mancinelli L. et al, (2020), VASCULAR PHARMACOLOGY, 132

Myocardial overexpression of ANKRD1 causes sinus venosus defects and progressive diastolic dysfunction.

Journal article

Piroddi N. et al, (2020), Cardiovasc Res, 116, 1458 - 1472

Myocardial redox state predicts 3-year mortality in patients undergoing cardiac surgery: the ox-HVF study

Conference paper

Kotanidis C. et al, (2019), EUROPEAN HEART JOURNAL, 40, 3886 - 3886

Novel direct effects of SGLT2 inhibitor, Canagliflozin, on myocardial redox state in humans

Conference paper

Kondo H. et al, (2019), EUROPEAN HEART JOURNAL, 40, 3872 - 3872

Wnt5a contributes to human atherosclerosis via novel USP17 redox signalling

Conference paper

Akoumianakis I. et al, (2019), EUROPEAN HEART JOURNAL, 40, 1660 - 1660

MicroRNA-34a modulates vascular calcification

Conference paper

Badi I. et al, (2018), VASCULAR PHARMACOLOGY, 103, 62 - 62

MicroRNA-34a role in vascular calcification

Conference paper

Badi I. et al, (2016), CARDIOVASCULAR RESEARCH, 111, S58 - S58

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