Publications — Radcliffe Department of Medicine

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Myocardial overexpression of ANKRD1 causes sinus venosus defects and progressive diastolic dysfunction.

Piroddi N. et al, (2020), Cardiovasc Res, 116, 1458 - 1472

The microRNA-34a-Induced Senescence-Associated Secretory Phenotype (SASP) Favors Vascular Smooth Muscle Cells Calcification.

Zuccolo E. et al, (2020), Int J Mol Sci, 21

Insulin-induced vascular redox dysregulation in human atherosclerosis is ameliorated by dipeptidyl peptidase 4 inhibition.

Akoumianakis I. et al, (2020), Sci Transl Med, 12

Myocardial redox state predicts 3-year mortality in patients undergoing cardiac surgery: the ox-HVF study

Kotanidis C. et al, (2019), EUROPEAN HEART JOURNAL, 40, 3886 - 3886

Novel direct effects of SGLT2 inhibitor, Canagliflozin, on myocardial redox state in humans

Kondo H. et al, (2019), EUROPEAN HEART JOURNAL, 40, 3872 - 3872

Wnt5a contributes to human atherosclerosis via novel USP17 redox signalling

Akoumianakis I. et al, (2019), EUROPEAN HEART JOURNAL, 40, 1660 - 1660

Adipose tissue-derived WNT5A regulates vascular redox signaling in obesity via USP17/RAC1-mediated activation of NADPH oxidases.

Akoumianakis I. et al, (2019), Sci Transl Med, 11

The curious case of dermal fibroblasts: cell identity loss may be a mechanism underlying cardiovascular aging.

Badi I., (2019), Cardiovasc Res, 115, e24 - e25

miR-34a Promotes Vascular Smooth Muscle Cell Calcification by Downregulating SIRT1 (Sirtuin 1) and Axl (AXL Receptor Tyrosine Kinase).

Badi I. et al, (2018), Arterioscler Thromb Vasc Biol, 38, 2079 - 2090

MicroRNA-34a modulates vascular calcification

Badi I. et al, (2018), VASCULAR PHARMACOLOGY, 103, 62 - 62

Non-oxidizable HMGB1 induces cardiac fibroblasts migration via CXCR4 in a CXCL12-independent manner and worsens tissue remodeling after myocardial infarction.

Di Maggio S. et al, (2017), Biochim Biophys Acta Mol Basis Dis, 1863, 2693 - 2704

DYSREGULATED EXPRESSION OF ANKYRIN REPEAT DOMAIN 1 IN THE DEVELOPING MYOCARDIUM CAUSES ANOMALOUS VENOUS RETURN AND MORPHOGENETIC DEFECTS BY IMPAIRING CARDIAC REMODELLING

Parolini C. et al, (2017), ATHEROSCLEROSIS, 263, E157 - E157

MicroRNA-34a role in vascular calcification

Badi I. et al, (2016), CARDIOVASCULAR RESEARCH, 111, S58 - S58

MicroRNA-34a Induces Vascular Smooth Muscle Cells Senescence by SIRT1 Downregulation and Promotes the Expression of Age-Associated Pro-inflammatory Secretory Factors.

Badi I. et al, (2015), J Gerontol A Biol Sci Med Sci, 70, 1304 - 1311

The mitochondrial lncRNA ASncmtRNA-2 is induced in aging and replicative senescence in Endothelial Cells.

Bianchessi V. et al, (2015), J Mol Cell Cardiol, 81, 62 - 70

A large permissive regulatory domain exclusively controls Tbx3 expression in the cardiac conduction system.

van Weerd JH. et al, (2014), Circ Res, 115, 432 - 441

Intracellular ANKRD1 protein levels are regulated by 26S proteasome-mediated degradation.

Badi I. et al, (2009), FEBS Lett, 583, 2486 - 2492

Transcriptional deregulation and a missense mutation define ANKRD1 as a candidate gene for total anomalous pulmonary venous return.

Cinquetti R. et al, (2008), Hum Mutat, 29, 468 - 474

Include forthcoming?