Publications — Radcliffe Department of Medicine

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Comprehensive epigenomic profiling reveals the extent of disease-specific chromatin states and informs target discovery in ankylosing spondylitis

Brown AC. et al, (2023), Cell Genomics, 100306 - 100306

Chromosome conformation capture approaches to investigate 3D genome architecture in Ankylosing Spondylitis.

Davidson C. et al, (2023), Front Genet, 14

FUNCTIONAL GENOMICS INVESTIGATION OF THE ANKYLOSING SPONDYLITIS ASSOCIATED LOCUS RUNX3

Cohen C. et al, (2022), ANNALS OF THE RHEUMATIC DISEASES, 81, 231 - 231

Functional Genomic Analysis of a RUNX3 Polymorphism Associated With Ankylosing Spondylitis.

Vecellio M. et al, (2021), Arthritis Rheumatol, 73, 980 - 990

INVESTIGATING THE ANKYLOSING SPONDYLITIS-ASSOCIATED REGULATORY SNPS AT THE RUNX3 LOCUS WITH A FUNCTIONAL GENOMICS APPROACH

Vecellio M. et al, (2021), ANNALS OF THE RHEUMATIC DISEASES, 80, 411 - 411

COMPREHENSIVE EPIGENOMIC PROFILING REVEALS DISEASE-SPECIFIC CHROMATIN STATES IN ANKYLOSING SPONDYLITIS

Cohen CJ. et al, (2021), CLINICAL AND EXPERIMENTAL RHEUMATOLOGY, 39, 1155 - 1155

Disruption of c-MYC Binding and Chromosomal Looping Involving Genetic Variants Associated With Ankylosing Spondylitis Upstream of the RUNX3 Promoter.

Cohen CJ. et al, (2021), Front Genet, 12

Perspectives on the Genetic Associations of Ankylosing Spondylitis.

Wordsworth BP. et al, (2021), Front Immunol, 12

A RUNX3 enhancer polymorphism associated with ankylosing spondylitis influences recruitment of Interferon Regulatory Factor 5 and factors of the Nucleosome Remodelling Deacetylase Complex in CD8+ T-cells

Vecellio M. et al, (2019)

The severity of ankylosing spondylitis and responses to anti-tumour necrosis factor biologics are not influenced by the tumour necrosis factor receptor polymorphism incriminated in multiple sclerosis.

Watts L. et al, (2019), Genes Immun, 20, 167 - 171

Quantifying the genetic risk for the development of axial spondyloarthropathy: could this become a diagnostic tool?

Wordsworth BP. et al, (2018), Curr Opin Rheumatol, 30, 319 - 323

INVESTIGATING THE REGULATORY SNPS AT THE RUNX3 LOCUS ASSOCIATED WITH ANKYLOSING SPONDYLITIS

Vecellio ML. et al, (2018), ANNALS OF THE RHEUMATIC DISEASES, 77, 158 - 158

Evidence for a second ankylosing spondylitis-associated RUNX3 regulatory polymorphism.

Vecellio M. et al, (2018), RMD Open, 4

EXPLORING THE ANKYLOSING SPONDYLITIS-ASSOCIATED REGULATORY SNPs AT THE RUNX3 LOCUS

Vecellio M. et al, (2018), CLINICAL AND EXPERIMENTAL RHEUMATOLOGY, 36, 734 - 734

RUNX3 and T-Bet in Immunopathogenesis of Ankylosing Spondylitis-Novel Targets for Therapy?

Vecellio M. et al, (2018), Front Immunol, 9

Investigation of a possible extended risk haplotype in the IL23R region associated with ankylosing spondylitis.

Roberts AR. et al, (2017), Genes Immun, 18, 105 - 108

An ankylosing spondylitis-associated genetic variant in the IL23R-IL12RB2 intergenic region modulates enhancer activity and is associated with increased Th1-cell differentiation.

Roberts AR. et al, (2016), Ann Rheum Dis, 75, 2150 - 2156

The genetic association of RUNX3 with ankylosing spondylitis can be explained by allele-specific effects on IRF4 recruitment that alter gene expression.

Vecellio M. et al, (2016), Ann Rheum Dis, 75, 1534 - 1540

THE ASSOCIATION OF A COMMON FUNCTIONAL POLYMORPHISM IN THE TUMOUR NECROSIS FACTOR RECEPTOR 1 GENE (TNFRSF1A) AND DISEASE SEVERITY IN ANKYLOSING SPONDYLITIS

Watts L. et al, (2016), RHEUMATOLOGY, 55, 150 - 150

A CCR6 variant strongly associated with rheumatoid arthritis in two populations is not associated with ankylosing spondylitis.

Cohen CJ. et al, (2013), Rheumatol Int, 33, 2443 - 2444

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