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Search results (64)

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Human MASLD is a diurnal disease driven by multisystem insulin resistance and reduced insulin availability at night.

Marjot T. et al, (2026), Cell Metab, 38, 474 - 492.e6

Functional Characterization of Glucokinase Variants to Aid Clinical Interpretation of Monogenic Diabetes.

Rajesh V. et al, (2025), Int J Mol Sci, 27

Oxidation of dietary linoleate occurs to a greater extent than dietary palmitate in vivo in humans.

Parry SA. et al, (2021), Clin Nutr, 40, 1108 - 1114

Electrophysiological properties of human beta-cell lines EndoC-βH1 and -βH2 conform with human beta-cells.

Hastoy B. et al, (2018), Sci Rep, 8

Type 2 diabetes risk alleles in PAM impact insulin release from human pancreatic β-cells.

Thomsen SK. et al, (2018), Nat Genet, 50, 1122 - 1131

Integration of human pancreatic islet genomic data refines regulatory mechanisms at Type 2 Diabetes susceptibility loci.

Thurner M. et al, (2018), Elife, 7

Genes Associated with Pancreas Development and Function Maintain Open Chromatin in iPSCs Generated from Human Pancreatic Beta Cells.

Thurner M. et al, (2017), Stem Cell Reports, 9, 1395 - 1405

Effect of Normothermic Machine Perfusion on Outcomes after Steatotic Liver Transplantation: A First Assessment

Ceresa CDL. et al, (2017), TRANSPLANTATION, 101, 161 - 162

A HISTOLOGICAL AND BIOCHEMICAL ASSESSMENT OF STEATOTIC LIVERS UNDERGOING NORMOTHERMIC MACHINE PERFUSION

Ceresa C. et al, (2017), TRANSPLANT INTERNATIONAL, 30, 43 - 43

Systematic Functional Characterization of Candidate Causal Genes for Type 2 Diabetes Risk Variants.

Thomsen SK. et al, (2016), Diabetes, 65, 3805 - 3811

Insights into islet development and biology through characterization of a human iPSC-derived endocrine pancreas model.

van de Bunt M. et al, (2016), Islets, 8, 83 - 95

Loss-of-Function Mutations in the Cell-Cycle Control Gene CDKN2A Impact on Glucose Homeostasis in Humans.

Pal A. et al, (2016), Diabetes, 65, 527 - 533

Type 2 Diabetes Risk Variants in PAM Reduce Chromogranin A-mediated Insulin Availability and Secretion from Beta Cells

Raimondo A. et al, (2016), DIABETES, 65, A52 - A52

Prioritising causal genes at type 2 diabetes risk loci through high-throughput screening for human beta cell dysfunction

Thomsen SK. et al, (2016), DIABETOLOGIA, 59, S86 - S87

Population whole-genome bisulfite sequencing across two tissues highlights the environment as the principal source of human methylome variation.

Busche S. et al, (2015), Genome Biol, 16

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