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Search results (11)

« Back to Publications

Transcriptomic Analysis of Inflammatory Cardiomyopathy Identifies Molecular Signatures of Disease and Informs in silico Prediction of a Network-Based Rationale for Therapy.

Singh K. et al, (2021), Front Immunol, 12

knottin scaffold directs the CXC-chemokine-binding specificity of tick evasins.

Lee AW. et al, (2019), J Biol Chem, 294, 11199 - 11212

The N-terminal domain of a tick evasin is critical for chemokine binding and neutralization and confers specific binding activity to other evasins.

Eaton JRO. et al, (2018), J Biol Chem, 293, 6134 - 6146

Genetically engineered two-warhead evasins provide a method to achieve precision targeting of disease-relevant chemokine subsets.

Alenazi Y. et al, (2018), Sci Rep, 8

Yeast surface display identifies a family of evasins from ticks with novel polyvalent CC chemokine-binding activities.

Singh K. et al, (2017), Sci Rep, 7

Corrigendum: Genetic affinities of the Jewish populations of India.

Chaubey G. et al, (2016), Sci Rep, 6

Genetic affinities of the Jewish populations of India.

Chaubey G. et al, (2016), Sci Rep, 6

Osteopontin deficiency affects imiquimod-induced psoriasis-like murine skin inflammation and lymphocyte distribution in skin, draining lymph nodes and spleen.

Frenzel DF. et al, (2015), Exp Dermatol, 24, 305 - 307

Unravelling the distinct strains of Tharu ancestry.

Chaubey G. et al, (2014), Eur J Hum Genet, 22, 1404 - 1412

Reduction of CD18 promotes expansion of inflammatory γδ T cells collaborating with CD4+ T cells in chronic murine psoriasiform dermatitis.

Gatzka M. et al, (2013), J Immunol, 191, 5477 - 5488

Reduced CD18 levels drive regulatory T cell conversion into Th17 cells in the CD18hypo PL/J mouse model of psoriasis.

Singh K. et al, (2013), J Immunol, 190, 2544 - 2553