{ "items": [ "\n\n
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\n A year in the life of the EU-CardioRNA COST action: CA17129 catalysing transcriptomics research in cardiovascular disease\n

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\n \n Journal article\n \n

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Robinson EL. et al, (2020), Non-coding RNA, 6

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\n Nitric Oxide and Mechano-Electrical Transduction in Cardiomyocytes.\n

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\n \n Journal article\n \n

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Boycott HE. et al, (2020), Front Physiol, 11

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\n The giant titin: how to evaluate its role in cardiomyopathies.\n

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\n \n Journal article\n \n

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Azad A. et al, (2019), J Muscle Res Cell Motil, 40, 159 - 167

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\n HspB1 phosphorylation regulates its intramolecular dynamics and mechanosensitive molecular chaperone interaction with filamin C.\n

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\n \n Journal article\n \n

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Collier MP. et al, (2019), Sci Adv, 5

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\n Genetics of and pathogenic mechanisms in arrhythmogenic right ventricular cardiomyopathy.\n

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\n \n Journal article\n \n

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Vimalanathan AK. et al, (2018), Biophys Rev, 10, 973 - 982

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\n Mutant Muscle LIM Protein C58G causes cardiomyopathy through protein depletion.\n

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\n \n Journal article\n \n

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Ehsan M. et al, (2018), J Mol Cell Cardiol, 121, 287 - 296

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\n Non-sarcomeric causes of heart failure.\n

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\n \n Journal article\n \n

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Gehmlich K. and Ehler E., (2018), Biophys Rev, 10, 943 - 947

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\n RNAseq reveals mechanisms of cardiomyopathy in Mlp-C58G knock-in mice\n

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\n \n Conference paper\n \n

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Ehsan M. et al, (2018), JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 120, 53 - 53

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\n Age- and strain-related aberrant Ca2+ release is associated with sudden cardiac death in the ACTC E99K mouse model of hypertrophic cardiomyopathy.\n

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\n \n Journal article\n \n

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Rowlands CT. et al, (2017), Am J Physiol Heart Circ Physiol, 313, H1213 - H1226

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\n Mammalian \u03b32 AMPK regulates intrinsic heart rate.\n

\n \n \n

\n \n Journal article\n \n

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Yavari A. et al, (2017), Nat Commun, 8

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\n When signalling goes wrong: pathogenic variants in structural and signalling proteins causing cardiomyopathies.\n

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\n \n Journal article\n \n

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Ehsan M. et al, (2017), J Muscle Res Cell Motil, 38, 303 - 316

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\n HSP27 phosphorylation regulates a mechanosensitive interaction with filamin C\n

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\n \n Conference paper\n \n

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Collier MP. et al, (2017), EUROPEAN BIOPHYSICS JOURNAL WITH BIOPHYSICS LETTERS, 46, S244 - S244

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\n Insights into disease mechanisms of cardiomyopathy from the Csrp3 C58G mouse model\n

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\n \n Conference paper\n \n

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Ehsan M. et al, (2017), JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 109, 11 - 11

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\n Insights into disease mechanisms of cardiomyopathy in the presence of the CSRP3 C58G mutation\n

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\n \n Poster\n \n

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Ehsan M. et al, (2017), EUROPEAN JOURNAL OF HEART FAILURE, 19, 341 - 342

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\n Evidence for Chaperone Function in Mechanosensation\n

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\n \n Journal article\n \n

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Collier M. et al, (2017), BIOPHYSICAL JOURNAL, 112, 491A - 491A

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\n Aberrant developmental titin splicing and dysregulated sarcomere length in Thymosin \u03b24 knockout mice.\n

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\n \n Journal article\n \n

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Smart N. et al, (2017), J Mol Cell Cardiol, 102, 94 - 107

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\n Combination of Whole Genome Sequencing, Linkage, and Functional Studies Implicates a Missense Mutation in Titin as a Cause of Autosomal Dominant Cardiomyopathy With Features of Left Ventricular Noncompaction.\n

\n \n \n

\n \n Journal article\n \n

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Hastings R. et al, (2016), Circ Cardiovasc Genet, 9, 426 - 435

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\n MLP and CARP are linked to chronic PKC\u03b1 signalling in dilated cardiomyopathy.\n

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\n \n Journal article\n \n

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Lange S. et al, (2016), Nat Commun, 7

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\n Heterozygous plakoglobin deficiency results in increased biventricular beta-catenin expression\n

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\n \n Conference paper\n \n

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Mak JKC. et al, (2015), EUROPEAN HEART JOURNAL, 36, 215 - 215

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\n PLAKOGLOBIN DEFICIENCY LEADS TO INCREASED BIVENTRICULAR BETA-CATENIN EXPRESSION IN THE MURINE HEART\n

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\n \n Conference paper\n \n

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Mak JKC. et al, (2015), HEART, 101, A124 - A124

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